Publications

Akt regulates L-type Ca2+ channel activity by modulating Cavalpha1 protein stability.

Catalucci D

J Cell Biol. 184(6):923-33. doi: 10.1083/jcb.200805063

The knockout of miR-143 and -145 alters smooth muscle cell maintenance and vascular homeostasis in mice: correlates with human disease.

Elia L

Cell Death Differ. 2009 Oct 9;16(12):1590-8. doi: 10.1038/cdd.2009.153

Reciprocal regulation of microRNA-1 and insulin-like growth factor-1 signal transduction cascade in cardiac and skeletal muscle in physiological and pathological conditions.

Elia L

Circulation. 2009 Nov 23;120(23):2377-85. doi: 10.1161/CIRCULATIONAHA.109.879429

MicroRNA control of podosome formation in vascular smooth muscle cells in vivo and in vitro.

Quintavalle M

J Cell Biol. 2010 Mar 29;189(1):13-22. doi: 10.1083/jcb.200912096

MTORC1 regulates cardiac function and myocyte survival through 4E-BP1 inhibition in mice.

Zhang D

J Clin Invest. 2010 Jul 19;120(8):2805-16. doi: 10.1172/JCI43008

Circulating miR-29a, among other up-regulated microRNAs, is the only biomarker for both hypertrophy and fibrosis in patients with hypertrophic cardiomyopathy.

Roncarati R

J Am Coll Cardiol. 2013 Oct 23;63(9):920-7. doi: 10.1016/j.jacc.2013.09.041

Genome-wide analysis of histone marks identifying an epigenetic signature of promoters and enhancers underlying cardiac hypertrophy.

Papait R

Proc Natl Acad Sci U S A. 2013 Nov 27;110(50):20164-9. doi: 10.1073/pnas.1315155110

Adeno-associated virus-mediated CASQ2 delivery rescues phenotypic alterations in a patient-specific model of recessive catecholaminergic polymorphic ventricular tachycardia.

Lodola F

Cell Death Dis. 2016 Oct 6;7(10):e2393. doi: 10.1038/cddis.2016.304

Histone Methyltransferase G9a Is Required for Cardiomyocyte Homeostasis and Hypertrophy.

Papait R

Circulation. 2017 Aug 4;136(13):1233-1246. doi: 10.1161/CIRCULATIONAHA.117.028561

The K219T-Lamin mutation induces conduction defects through epigenetic inhibition of SCN5A in human cardiac laminopathy.

Salvarani N

Nat Commun. 2019 May 22;10(1):2267. doi: 10.1038/s41467-019-09929-w